Can anyone explain in laymans terms what is going on during "less-than-full" apnea? I have searched and searched allready.
Taken from another thread:
Now, this is what I think I get: For clarity, I consider empty-lung apnea first.
- We have less oxygen overall initially.
- DR is stronger because of fast CO2 buildup in beginning
- CO2 i high because it cannot move from bloodstream to lungs (not much anyway)
- later in the (static emptylung) dive it is easier to BO because......???
Wouldn't the higher CO2 make it "safer" because of urge to breeth? Or is CO2 only high until a point when vasoconstriction is strong, and CO2 "stops" building up fast, and only builds up from the core, very slowly.
Could someone please explain it, starting with simple, straight forward information to begin with?
Thanks
Taken from another thread:
There's something I don't understand well, even though I have read a very scientific explanation before (perhaps too difficult for me).
Is it easier to BO when doing less-than-full/exhale/FRC breathholds???
Some say it is safer, fx Sebastion Murat in this video because of stronger DR as I understands it.
Others say it is easier to BO along the lines that less CO2 is building up, and therefore it is easier to reach blackout.
Now I know Sebastion is very much into benefits of exhaling in depth-diving. So just for a second, to try separate different contributing factors, how about just a regular static? It seems it comes down to STRONG DR vs. CO2 BUILDUP.
Are there different believes and experiences about wich is "safer"?
Or is there perhaps like someone mentioned a "sweetspot" in exhaling, an optimal degree/amount of both DR and CO2?
Now, this is what I think I get: For clarity, I consider empty-lung apnea first.
- We have less oxygen overall initially.
- DR is stronger because of fast CO2 buildup in beginning
- CO2 i high because it cannot move from bloodstream to lungs (not much anyway)
- later in the (static emptylung) dive it is easier to BO because......???
Wouldn't the higher CO2 make it "safer" because of urge to breeth? Or is CO2 only high until a point when vasoconstriction is strong, and CO2 "stops" building up fast, and only builds up from the core, very slowly.
Could someone please explain it, starting with simple, straight forward information to begin with?
Thanks
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