Mullins said:
I may be off-track here, but my understanding is that RV is determined by the limit to which the ribcage/diaphragm can be compressed by ambient pressure before resistance increases to the point where the pressure differential between the space in the lungs and the ambient pressure has to be equalised in another way - by liquid first filling the alveoli forming the walls of the lung cavity and then forcing its way through them (causing damage) into the space itself.
I don't understand RV to be an attribute of the lung tissue itself (please correct me if this is wrong). The term 'RV' as it is commonly used seems to me to mean "lung volume following forceful exhale without other mechanical aid" and as such is reliant on the force supplied by the breathing muscles as well as the flexibility of the diaphragm/ribcage. Given adequate mechanical force (over and above that supplied solely by the breathing muscles) RV for everybody would be zero. Given sufficient bloodshift even in a thoracic inflexible person, RV would also be zero. Obviously this would mean a nasty injury in both cases because nobody is that flexible/nobody's alveolic can take that much engorgement, it's just my way of suggesting RV is not an absolute value unless you add the above condition.
RV depends of pulmonary and extra-pulmonary variables. As you pointed out, a person with a very strong diaphragm and very flexible thorax has a lower RV than a person without that conditions, but there is also intra-alveolar pressure that depends on surfactant, compliance and patency of the airway that communicates with it, this is refered as the Times Constant, so there is slow and fast alveoli (to empty and to fill). Persons with problems like asthma can have a increased RV during crisis, because the alveoli can't get empty on a normal exhalation.
I think is very difficult to reach values below RV, in fact RV can vary within the same individual. Blood-shift help you out to reach a lower RV without pulmonary collapse. When you pack the VC increases and RV decreases, but this is just an static measure. Nobody has measured Lung volumes during real immersion, and in the other hands, we don't know the long time effects of frequent lung packing, if, and this is purely theoretical, after frequent lung packing we loss elastic recoil of the lung, our RV can increase due to this practice.
naiad said:
Would reverse packing reduce the risk of squeeze? If packing increases RV, then reverse packing would surely reduce it?
Just an idea...
At least in theory it could be true. I´m living far away from places to practice real CW training, so in the 2 or 3 times in the year that can get to deep places I was posing my self in risk for immersion pulmonary edema (I got once to a ridiculous depth of 30 mts), so I started an slow and progressive training with reverse packing in the pool, and until now it has worked, but I think is more due to improve in chest flexibility, but can't discard some kind of thickening in my blood vessels.
Cebaztian said:
My hypothesis is that things break around the trachea area and that fluids goes down into lung and causes near drowning symptoms (described).
I do believe, that in some cases the blood comes from the trachea, but for this blood to produce low oxygen saturation it has to be A LOT. I do Bronchoalveolar lavages to patients and some times I put and amount of saline solution of around 300-400 ml. without oxygenation problems. So the amount of blood to flood the lungs and produce near drowning has to be huge, more than just a spit with traces of blood.
As Sebastian said, this is quite confusing, because we don't have enough information, his suggestion to report it to DAN is a good start.
But after the information I had, contractions and excessive lung volumes prior to immersion are the main causes (just my opinion) the other is the fast progression without training on extreme volumes changes.