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I made these statements earlier, but after some more study, I have discovered how wrong I was. When I originally studied my wife’s Human Anatomy & Physiology textbook I read how steep the saturation curve rises with changes in PO2 (Oxygen Particle pressure) and how a change in ph shifts the whole curve to the left or right. What I failed to see and understand is the curve rises steeply at low PO2. Normal alveolar pressure is 100 mm Hg. At this rate hemoglobin saturation is already 97.5%. From this point the curve rises very slowly to where it takes a PO2 of 250 mm Hg before it finally reaches 100% saturation.I think the acidity blood is crucial. It’s the key to getting as much O2 in the blood as possible. More O2 in the blood will have more of a positive (direct) effect on long statics than any O2 loading of body tissue. In a normal breath we are only drawing out fraction of the available O2 from the air in our lungs. The reason why is because we easily reach our blood saturation max. Raising the saturation max point, I believe, would have the most direct effect.
So in other words, instead of trying to get more O2 in, I think we should focus on trying to slow the amount going out.